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Tissue Models

Compound K suppresses epidermal aging induced by IL-17A treatment and UVB irradiation

  • TR Number: 1078
  • Authors: A Young Kim , Ji-Yong Jung , Donghyun Kim , Jeong Hun Cho , Yong Deog Hong, Hyoung-June Kim, Si-Young Cho
  • Keywords: EpiDerm, EPI-201, skin aging, inflammaging, IL-17a, UVB, Compound K, ginsenoside, anti-inflammatory, accelerated aging model, epidermal thickness, Ki-67, filaggrin, Cytokeratin K10, CK10, collagen 17A1, IL-8, IL-6, TNF, GM-CSF, H&E, neutralizing antibody  
  • Materials Tested: IL-17a, UVB, Compound K, ginsenoside
  • Link to Article: https://www.sciencedirect.com/science/article/pii/S1226845325000284

We evaluated the anti-accelerated epidermal skin aging effects of compound K (CK) on keratinocytes using IL-17A and ultraviolet B. CK restored epidermal functions and reduced the induction of pro-inflammatory cytokines by inhibiting NF-κB and p38 MAPK activation. CK plays a significant role in controlling accelerated epidermal skin aging.